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[SalvatoreCorvus]

Can you guys please credit my account with points so I can still buy from this sale? @SalvatoreCorvus @Donvitosharkone

You can't use code and credit at the time.
And your credit withdrawal as to be validate by the admin not moderator

 

[SalvatoreCorvus]

I can’t find it

Please open a ticket to the customer service. The retatrutide item is sometimes not visible to new customers.

Customer service will add it to your shopping cart for you.

Contact Us - Driadashop.to HelpDesk

driadashop.helpline.to

 

[Donvitosharkone]

Can you guys please credit my account with points so I can still buy from this sale? @SalvatoreCorvus @Donvitosharkone

You have registered your request in the right spot.

Let’s see if we can make it full speed.

 

[TrenReplacementTherapy]

You have registered your request in the right spot.

Let’s see if we can make it full speed.

thanks a lot, Don!

 

[Mr_Hybrid]

Vorrei acquistare sibutramina ma ho sentito tante brutte storie di ictus e infarti io ho 45 anni e sono sanissimo però mi preoccupa un po', mi chiedo perché non avete inserito FENTETMINA 75 che è ancora legale ed in commercio in alcune nazioni mentali sibutramina è fuori produzione dal 2010.

 

[natondica]

Why you can't ship to Poland?

 

[Driadashop]

I can’t find it

Please ask to add it to your order guys from the support https://driadashop.helpline.to/en-US/new-ticket

 

[Driadashop]

Can you guys please credit my account with points so I can still buy from this sale? @SalvatoreCorvus @Donvitosharkone

Hi! Tell please what didn't you get points for?

 

[Driadashop]

Why you can't ship to Poland?

Hello! Unfortunately, there are no deliveries to your region, but you can place an order in one of the regions where delivery is possible, for example, if you have friends or relatives there. Also, pickup points are available in some regions, maybe you can pick up the parcel.

 

[Driadashop]

Vorrei acquistare sibutramina ma ho sentito tante brutte storie di ictus e infarti io ho 45 anni e sono sanissimo però mi preoccupa un po', mi chiedo perché non avete inserito FENTETMINA 75 che è ancora legale ed in commercio in alcune nazioni mentali sibutramina è fuori produzione dal 2010.

Ciao! Si prega di contattare il servizio di consulenza the PEDs Father https://thepedsfather.taplink.ws

 

[TrenReplacementTherapy]

Hi! Tell please what didn't you get points for?

I am trying to withdraw my points from this forum to my driada account and it says pending for multiple days

 

[Daniil Nuerra]

In this field, HPLC is the standard analytical method used to verify the concentration of the active ingredient in oil-based products.

We test every batch of raw material by HPLC before production begins to ensure it meets our quality standards.

In addition, the finished products are independently tested by our customers through blind testing at third-party laboratories. To encourage independent verification and transparency, we support blind testing by offering €200 in store credit to customers who submit eligible laboratory reports.

As a result, we now have hundreds of independent blind test results confirming the quality and dosage of our products.

You can view these laboratory reports in the Reviews section by opening any product: https://peds.to/reviews/

I meant not the Q/Q test, more of full panel of solvents/oils etc, what is "Testos" exactly is, this oils or is there EO in it...

 

[TrenReplacementTherapy]

I meant not the Q/Q test, more of full panel of solvents/oils etc, what is "Testos" exactly is, this oils or is there EO in it...

They have a page search up driada carrier oils they made a post about it on the site, yes unfortunately theres EO in it I really really wish they changed it but for me it doesnt raise C reactive protein much, still this is my only issue with driada

 

[Daniil Nuerra]

They have a page search up driada carrier oils they made a post about it on the site, yes unfortunately theres EO in it I really really wish they changed it but for me it doesnt raise C reactive protein much, still this is my only issue with driada

Cause for me, unfortunately D.oils raises CRP ((, No lumps, no PIP if technique is correct, so by the "feelings" perfect, but the analisys shows not the best results, even on 150mg/4days of Test E/C it raises to 5 mg/l (Which is visibly elevated and its not HS, just basic CRP analysis). Yep other parameters is good, but I want to know more of the reson, if its anywhere Screening of oils and Ill see that it contains high EO - I would have a data to think of... Im on everlasting TRT for now, so its important, I understand some CRP throughout the cycle for some month but for years....

 

[TrenReplacementTherapy]

Cause for me, unfortunately D.oils raises CRP ((, No lumps, no PIP if technique is correct, so by the "feelings" perfect, but the analisys shows not the best results, even on 150mg/4days of Test E/C it raises to 5 mg/l (Which is visibly elevated and its not HS, just basic CRP analysis). Yep other parameters is good, but I want to know more of the reson, if its anywhere Screening of oils and Ill see that it contains high EO - I would have a data to think of... Im on everlasting TRT for now, so its important, I understand some CRP throughout the cycle for some month but for years....

Yeah probably better to use another lab in that case I really wish driada stopped using this shit

 

[Driadashop]

Yeah probably better to use another lab in that case I really wish driada stopped using this shit

Regarding the connection between elevated CRP and ethyl oleate in oils — we've discussed this topic many times, and not only on this forum. Let's break it down properly. The question isn't whether someone measured something incorrectly, but how the cause is being interpreted. And this is exactly where it's important not to jump to conclusions.

Where the whole EO raises CRP conversation came from
This is primarily a forum narrative, not a scientific one. It's been circulating in communities (Evolutionary, MESO) since at least 2016. One of the more recent triggers was a video from a "reputable YouTuber" comparing synthetic and natural carrier oils, where it was claimed that synthetic solvents like EO cause systemic inflammation. It's after videos like these that people come to forums asking "is EO dangerous?" From there, the classic pattern kicks in: influencer + personal observations of a few individuals → generalization into "EO raises CRP in everyone."

What more plausibly explains the CRP increase
The toxicity of EO is considered low, and it's used as a carrier in pharmaceuticals. There are also no direct studies showing that EO raises CRP in humans. So there's no real scientific basis behind this specific claim.

Beyond that, CRP rises for a huge number of reasons: testosterone itself and estradiol levels, visceral fat, training stress, diet, hematocrit, hidden inflammatory processes. And if you don't run a genuinely proper experiment, the true cause can't be pinned down. In that situation, EO becomes a convenient scapegoat — simply because it's the most visible non-standard component in the blend.

And for that matter, practically all oils contain BB and BA, which have a greater irritant potential. Any component that causes local tissue irritation at the injection site increases local inflammation and the release of inflammatory cytokines (IL-6), and consequently the liver ramps up CRP production. CRP doesn't distinguish what caused the inflammation — it responds to the inflammation itself.

For example, benzyl benzoate (BB) has higher toxicity and irritant potential than EO, yet it's used by practically every brand — and for some reason nobody in the community talks about it. And I'll venture a guess as to why: that well-known blogger used the EO accusation as a marketing move to produce a "special natural product" and gain a competitive edge (and it has to be said, he succeeded at it). But since abandoning BB is practically impossible in practice, he simply stayed quiet about it.

How to detect the reason
The only way to establish the cause is a clean experiment. Remove the oil carrier for a period, switch to a gel or oral testosterone, and retest CRP several times over time: at 2, 4, and 6 weeks. Keep your lifestyle stable throughout the whole period — don't change your diet or training in parallel, don't start or stop anything.

After it normalizes, reintroduce the same product and see whether CRP rises again. It's the reproducibility of the reaction that distinguishes causation from coincidence.

If CRP normalizes — that's a strong argument in favor of the carrier, and then it makes sense to change the form of administration.
If it stays elevated — then the cause lies elsewhere, and you need to dig deeper.

 

[TrenReplacementTherapy]

Regarding the connection between elevated CRP and ethyl oleate in oils — we've discussed this topic many times, and not only on this forum. Let's break it down properly. The question isn't whether someone measured something incorrectly, but how the cause is being interpreted. And this is exactly where it's important not to jump to conclusions.

Where the whole EO raises CRP conversation came from
This is primarily a forum narrative, not a scientific one. It's been circulating in communities (Evolutionary, MESO) since at least 2016. One of the more recent triggers was a video from a "reputable YouTuber" comparing synthetic and natural carrier oils, where it was claimed that synthetic solvents like EO cause systemic inflammation. It's after videos like these that people come to forums asking "is EO dangerous?" From there, the classic pattern kicks in: influencer + personal observations of a few individuals → generalization into "EO raises CRP in everyone."

What more plausibly explains the CRP increase
The toxicity of EO is considered low, and it's used as a carrier in pharmaceuticals. There are also no direct studies showing that EO raises CRP in humans. So there's no real scientific basis behind this specific claim.

Beyond that, CRP rises for a huge number of reasons: testosterone itself and estradiol levels, visceral fat, training stress, diet, hematocrit, hidden inflammatory processes. And if you don't run a genuinely proper experiment, the true cause can't be pinned down. In that situation, EO becomes a convenient scapegoat — simply because it's the most visible non-standard component in the blend.

And for that matter, practically all oils contain BB and BA, which have a greater irritant potential. Any component that causes local tissue irritation at the injection site increases local inflammation and the release of inflammatory cytokines (IL-6), and consequently the liver ramps up CRP production. CRP doesn't distinguish what caused the inflammation — it responds to the inflammation itself.

For example, benzyl benzoate (BB) has higher toxicity and irritant potential than EO, yet it's used by practically every brand — and for some reason nobody in the community talks about it. And I'll venture a guess as to why: that well-known blogger used the EO accusation as a marketing move to produce a "special natural product" and gain a competitive edge (and it has to be said, he succeeded at it). But since abandoning BB is practically impossible in practice, he simply stayed quiet about it.

How to detect the reason
The only way to establish the cause is a clean experiment. Remove the oil carrier for a period, switch to a gel or oral testosterone, and retest CRP several times over time: at 2, 4, and 6 weeks. Keep your lifestyle stable throughout the whole period — don't change your diet or training in parallel, don't start or stop anything.

After it normalizes, reintroduce the same product and see whether CRP rises again. It's the reproducibility of the reaction that distinguishes causation from coincidence.

If CRP normalizes — that's a strong argument in favor of the carrier, and then it makes sense to change the form of administration.
If it stays elevated — then the cause lies elsewhere, and you need to dig deeper.

I also think its very genetics dependant, the highest my crp has been was 2. Something on driada stuff (about 800 mg total) so this isnt really a dealbreaker for me.
From what I heard EO is only approved for use in Pharma injectables in Asia, not in the EU. Also please do not take my slight criticism as slander of the brand, I have found every product so far to be reliable.

 

[TrenReplacementTherapy]

I also think its very genetics dependant, the highest my crp has been was 2. Something on driada stuff (about 800 mg total) so this isnt really a dealbreaker for me.
From what I heard EO is only approved for use in Pharma injectables in Asia, not in the EU. Also please do not take my slight criticism as slander of the brand, I have found every product so far to be reliable.

It was 0.03 hs crp on 150 mg test e from driada

 

[Daniil Nuerra]

Regarding the connection between elevated CRP and ethyl oleate in oils — we've discussed this topic many times, and not only on this forum. Let's break it down properly. The question isn't whether someone measured something incorrectly, but how the cause is being interpreted. And this is exactly where it's important not to jump to conclusions.

Where the whole EO raises CRP conversation came from
This is primarily a forum narrative, not a scientific one. It's been circulating in communities (Evolutionary, MESO) since at least 2016. One of the more recent triggers was a video from a "reputable YouTuber" comparing synthetic and natural carrier oils, where it was claimed that synthetic solvents like EO cause systemic inflammation. It's after videos like these that people come to forums asking "is EO dangerous?" From there, the classic pattern kicks in: influencer + personal observations of a few individuals → generalization into "EO raises CRP in everyone."

What more plausibly explains the CRP increase
The toxicity of EO is considered low, and it's used as a carrier in pharmaceuticals. There are also no direct studies showing that EO raises CRP in humans. So there's no real scientific basis behind this specific claim.

Beyond that, CRP rises for a huge number of reasons: testosterone itself and estradiol levels, visceral fat, training stress, diet, hematocrit, hidden inflammatory processes. And if you don't run a genuinely proper experiment, the true cause can't be pinned down. In that situation, EO becomes a convenient scapegoat — simply because it's the most visible non-standard component in the blend.

And for that matter, practically all oils contain BB and BA, which have a greater irritant potential. Any component that causes local tissue irritation at the injection site increases local inflammation and the release of inflammatory cytokines (IL-6), and consequently the liver ramps up CRP production. CRP doesn't distinguish what caused the inflammation — it responds to the inflammation itself.

For example, benzyl benzoate (BB) has higher toxicity and irritant potential than EO, yet it's used by practically every brand — and for some reason nobody in the community talks about it. And I'll venture a guess as to why: that well-known blogger used the EO accusation as a marketing move to produce a "special natural product" and gain a competitive edge (and it has to be said, he succeeded at it). But since abandoning BB is practically impossible in practice, he simply stayed quiet about it.

How to detect the reason
The only way to establish the cause is a clean experiment. Remove the oil carrier for a period, switch to a gel or oral testosterone, and retest CRP several times over time: at 2, 4, and 6 weeks. Keep your lifestyle stable throughout the whole period — don't change your diet or training in parallel, don't start or stop anything.

After it normalizes, reintroduce the same product and see whether CRP rises again. It's the reproducibility of the reaction that distinguishes causation from coincidence.

If CRP normalizes — that's a strong argument in favor of the carrier, and then it makes sense to change the form of administration.
If it stays elevated — then the cause lies elsewhere, and you need to dig deeper.

Exactly what im doing. Stopped Testosterone, waiting some weeks on HCG to test...